Archive for July, 2011

Age related memory issues

Thursday, July 28th, 2011

Brain 2

I found this article today:  this isn’t any surprise to me as we’ve been working with improving people’s memory for many years:

  • insulin resistance (which we can definitely reverse) leads to dementia
  • B12/folate deficiency (not found in bloodtests) leads to lack of genetic protection and our brain can falter.  2000mg-4000mg daily for 3 weeks – if memory improves it can be early onset dementia
  • kinesiology – LEAP – works with working memory, hippocampus, parahippocampus and other momory centres.
  • microcurrent can reduce inflammation in the brain, which causes premature ageing.
  • Stem Enhance for regeneration of any tissue in the body which needs it – increases stem cell production release from the bone marrow.
When reading the following article, take care – chemical imbalances don’t mean drugs.  We don’t have a deficiency of effexor or paxil or valium.  We have nutrient deficiencies – B12, zinc, C, E, coenzyme Q10.  We can find and work with real deficiencies with real tests.
Madonna Guy ND
Wynnum, Brisbane, Australia
3348 6098 / 0417 643 849

The cause of age-related memory loss — that moment of “where did I leave my keys?” — has been found and reversed in monkeys, researchers announce.

By restoring a chemical imbalance in the brains of elderly monkeys, the study scientists transformed aging brain cells into young ones, capable of sustaining working memory, through which the brain is able to hold a thought for a short period of time.

Monkeys have long been used as a model for human brain studies, and so the results may also hold true for humans, though further research is needed to confirm that.

“This starts to change very early — by your thirties or so, you start to show decline in your working memory. This kind of memory is actually quite vulnerable,” said Carol Barnes, Director of the Evelyn F. McKnight Brain Institute at the University of Arizona, who was not involved in the study.

“What’s exciting about this study is the more you understand about how memory changes in different brain regions, the better we will be able to do something about it,” Barnes added.

Monkey brains
The researchers studied working memory in a group of monkeys separated into three life stages: Young monkeys (ages 7 and 9, or the equivalent of 21-27 in human years), middle-age monkeys (12 and 13, or 36-39 in human years), and older monkeys (17 and 21, or 51-63 in human years).

Using special tiny probes, the researchers watched single cells in each monkey’s prefrontal cortex (where working memories are formed and stored) as the monkeys performed a memory task; the monkeys had to remember the location of a treat in a computer simulation for 2.5 seconds, a time short enough for even the oldest monkeys to perform well. If they don’t do well at the test, the older monkeys get frustrated and won’t play anymore, study researcher Amy Arnsten of Yale University School of Medicine told LiveScience.

As expected, the memory circuit in the brains of older monkeys didn’t sustain activity as well as in younger monkeys, even though they still performed accurately at this short time scale. In longer trials, up to 5 seconds, they didn’t perform as well as the younger monkeys.

The researchers knew that a chemical called cyclic AMP played an important role in memory formation in the prefrontal cortex and that too much of the brain chemical could lead to forgetfulness in monkeys and humans alike.

To test if lowering cyclic AMP could help restore brain function in these monkeys, the researchers injected tiny amounts of drugs that block it in the vicinity of certain memory-holding neurons. Then, they had the monkeys redo the tests. The drugs improved the ability of these cells to hold a signal, though because the monkeys were already performing well on the tests, the researchers couldn’t see improvement on the actual tasks.

“If we restore the correct neurochemical environment, they were able to fire like young neurons,” Arnsten said. “It’s very easy for something to be impaired and not work right; you have to really understand what’s going on to get them to work again.”

Autism, ADHD, Aspergers, Bipolar, ODD: Are we being diagnosed to death?

Wednesday, July 27th, 2011

It’s been interested researching the origins of Psychiatric Terminology/Definitions/Diagnosis…  Have you or a loved one been diagnosed with a mental ‘disorder’ that can only be fixed by drugs ‘for the rest of your life?’.  Since the 1950’s new psychiatric terms have been ‘voted on’ by psychiatrists with links to drug companies.   They vote on the symptoms, what should and shouldn’t be included.  Diagnosis change depending on political input and $$$ put in from different Drug Companies.  In order to make the largest amount of $$$ they need to have a new beaut ‘illness’ to prescribe a newer (often repackaged) drug.  It’s a trillion dollar industry.

So many people believe that the diagnosis is lucky to have been found – thank goodness they’ve found a reason for my feeling low, losing my temper, having really bad days…  and there’s a drug to fix it.  Isn’t ‘feeling emotional’ part of being human?  We’re trying to suppress the very thing that separates us from animals!

There are over 70,000 known side effects of psychiatric drugs – everything from death (worst case scenario), dementia, rages, chronic depression, digestive disorders, further ‘mental illness’, non-verbal behaviours and more.

In the U.S.A. children are being screened (questionaires) filled out be children in over 43 states, without parental permission.  Over 50% of these questionaires that give a diagnosis of ‘high suicide risk’ or ‘pediatric biplar’ or ‘ADHD’ send parents to psychologists/psychiatrists who instantly prescribe drugs from the questionaire.

This is scary business.

Being on psychiatric drugs increases risk of child/teenage suicide by 65x.  Is that a risk worth taking?

Children on ritilan for concentration are well known to become addicts to stronger drugs as they get older, snorting ritilan for a ‘faster fix.’

What are we doing to our children?  It is becoming more and more acceptable to put our children (from 2 years old) onto medical drugs to suppress their childhood urges and to stop their ‘moodswings’ which are simply from being a child.

What can we do to help?

We have several specific programmes and solutions that support children:

  • underlying health issues? thyroid? iron levels? B12/folate?  liver function? hormonal issues?  Often we are whacked on a psychiatric drug so quickly that no medical doctor has even thought of taking a look at other health issues.
  • looking at nutrition.  What are we eating?  Are we absorbing our food?  Do we have a DPP4 enzyme deficiency in the gut causing food staples such as wheat and dairy to act like opioids (Drugs) in the body and brain?  We would also prescribe (if necessary, minerals, homeopathics, herbal combinations) depending on what’s going on.
  • is our gut functional? There is a massive gut-brain connection – if the gut is dysfunctional, the brain will struggle.  Are our kids starting the day with only carbohydrates (cereals/toast) which turn into sugar, raise blood sugar, and make them cranky/unable to concentrate?
  • Kinesiology/Microcurrent therapy: the combination is fabulous – it helps to integrate the brain and detox the body.

Call us today for an initial consultation to get you or your loved one started on the road ‘back to health’.
Madonna Guy ND
New Leaf Natural Therapies
Brisbane, Wynnum, Australia
3348 6098 / 0417 643 849

When Breastfeeding is NOT best – mums on anti-depressants!

Thursday, July 21st, 2011

When Breastfeeding Is NOT Best

Posted by danielle sullivan on June 27th, 2011 at 12:01 am
868023 81052427 300x220 When Breastfeeding Is NOT BestBottle feeding is the better alternative for some babies.

There is no disputing the many inherent health benefits that breastfeeding promotes, for both baby and mom, but in some cases it’s just not optimal. In some cases, it should be discouraged.

Stephanie Greene is one such case.

Greene’s six-week-old baby, Alexis was poisoned by her mom’s breast milk which was tainted with morphine. Now Stephanie Greene is facing a homicide charge and more than three dozen drug-related charges.

The NY Daily News reports that South Carolina medical examiner determined that the baby died from a lethal dose of morphine that was ingested through her mother’s breast milk:

“Doing toxicology tests and things like that, we wanted to make sure, and the coroner’s office wanted to make sure that we had done everything correctly and possible to make sure we understood fully what had happened to this child, how this child died and who was responsible for it.”

Greene allegedly used fake prescriptions to obtain the drugs. Later, she took pills and used patches containing morphine around the time baby Alexis was born. She reportedly took fentanyl, duragesic, morphene and hydrocodon, and had obtained the drugs at least 38 times, in less than two years at the same pharmacy.

In the cases of mothers who are so drug addicted, they are simply unable to keep their own bodies healthy and drug-free, it makes sense that bottle feeding should not only be encouraged, but mandated. But that also might leave a slippery slope for women who smoke, drink to excess, or consume an unhealthy diet.

From Madonna Guy ND: Obviously, whatever we ingest throughout pregnancy does go through to the baby.  Babies are born (on average) with over 200 carcinogenic substances in their umbilical cord blood.  The idea that they’re safe is antiquated and wrong.

We do ‘safe’ detoxification prior to our fertility programmes as well as throughout pregnancy and breastfeeding.  Taking care of your child is of the utmost importance.

No-one wants to bury a child.

New Leaf Natural Therapies
Wynnum Brisbane
3348 6098

Mums on Anti-depressants – links to autism

Thursday, July 21st, 2011

From Madonna:  this is an article from 21/7/11 published in the New York Times:

A preliminary but provocative new study finds women who take antidepressants during pregnancy have a moderately higher risk of having a child with autism, according to a paper published in the Archives of General Psychiatry.

Medicated Moms (Mums)

Use of prescription drugs in the first trimester of pregnancy:

  • Antidepressant use grew to about 7.5% of pregnant women in the 2006-2008 period, up from 5% in 2000-2002.
  • Until 1990, less than 1% of pregnant women used antidepressants in the first trimester.
  • Since 1976, use of all types of prescription drugs among pregnant women more than doubled.
  • About half of women reported that they took at least one prescription drug during pregnancy.

Source: Allen Mitchell, Slone Epidemiology Center at Boston University and colleagues; American Journal of Obstetrics and Gynecology.

Another study, published in the same issue of the journal and examining autism in pairs of identical and fraternal twins, finds that environmental factors play a greater role than previously believed in the development of autism, underscoring the need to understand nongenetic causes of autism.

The research on antidepressants and autism is thought to be the first to look for and identify such a link. Results indicated a doubling in risk of autism if the mother filled a prescription for antidepressants at any point in the year before delivery. The risk tripled if she filled the prescription during the first trimester of pregnancy.

The findings don’t speak to whether antidepressants cause autism, and the work needs to be replicated, the authors cautioned. The data, though, do indicate that the drugs have “possible adverse outcomes in children” and deserve further study, said Lisa Croen, first author on the study and an epidemiologist in the research division of Kaiser Permanente Northern California, the big managed-health plan.

“A lot of people might get a little worried about these findings and change something they’re doing—which they shouldn’t. It indicates to us that there’s more to look at,” said Dr. Croen, who also is an author on the twins study.

The researchers, sifting through medical records, identified 298 children diagnosed with autism or a similar disorder and looked back in time to the characteristics of the mothers. These children and mothers were compared with 1,507 children without autism and their mothers.

The relationship between autism in the child and the mother’s use of antidepressants—predominantly the category known as selective serotonin reuptake inhibitors, or SSRIs—remained even after researchers statistically accounted for the effects of other factors that might be related to either condition, such as maternal age, ethnicity and education, as well as baby birth weight and where the baby was born. In the twins study, a team including researchers from the University of California San Francisco Institute for Human Genetics, Kaiser Permanente and the California Department of Public Health identified sets of twins born in California between 1987 and 2004 in which at least one twin was diagnosed with autism or a related disorder. They conducted genetic testing on 192 twin pairs to determine whether they were identical or fraternal and recorded whether each individual qualified for an autism diagnosis.

Then they compared autism rates in fraternal twins versus identical twins, when one twin had it and also when both twins had it. If autism were a completely genetic disorder, both twins in each identical-twin pair would have it, the researchers figured. And if it were caused completely by environmental factors, the autism rates in fraternal twin pairs and identical twin pairs would be the same.

The results indicate that roughly half the risk of autism was accounted for by environmental factors—far more than detected in previous studies, according to Joachim Hallmayer, psychiatry professor at Stanford University School of Medicine and first author on the study. It “shows clearly that we have to take both environment and genes seriously, and we have to study much more the interactions between genes and environment,” Dr. Hallmayer said. Environmental factors shared by twins, particularly during the prenatal period and right after birth, may contribute to autism, he said.

In the antidepressant study, researchers tried teasing apart whether the mother’s mental state or the antidepressants were linked with autism. The results indicated an association with the treatment, not with the mother’s mental state.

If the pattern can be replicated in a broader population of children, the findings “will add to the growing list of cautions about exposing children and adolescents to medications without a very clear demonstrated need,” said John March, director of neurosciences medicine at the Duke Clinical Research Institute, who wasn’t involved in the study. Because of limited information in the medical records, researchers weren’t able to look at other important factors that might also affect fetal development such as ultrasounds and pain medicines, he added.

Previous research has shown that people with autism have female relatives with a greater likelihood of depression or anxiety. So what looks like a link between antidepressants and autism could actually be a genetic predisposition to this cluster of conditions, said Fred Volkmar, director of Yale University’s Child Study Center, who wasn’t involved in the current study.

Doctors and patients must weigh the risk of taking antidepressants in pregnancy against risks to the unborn child of untreated depression in the mother. A woman who is depressed may not eat regularly or keep prenatal checkups—possibly putting her baby at more risk than if she took antidepressants, said Mason Turner, Kaiser Permanente San Francisco’s chief of psychiatry.

From Madonna:

We can help with depression, post-natal depression, and autism…

Give us a call if you feel that anti-depressants may be causing an issue in your life, your baby or your pregnancy.

Madonna Guy ND
New Leaf Natural Therapies
3348 6098